Author: foxl

Well I made this Saturday, from lowcarb friends and other places online:

using the frozen cauli since I have a ton of it.

2 c. cauli
2 egg
2 c mozzarella

After microwaving, I mashed cauli with a potato masher, added egg and cheese, pressed into pan and sprinkled with ground fennel, oregano, and salt and pepper … baked 20 minat 400, and then topped with red sauce (LOTS of oregano and black pepper), then sauteed mushrooms and roasted strips of red pepper, and more mozzarella, and broiled till the cheese melted.

It was GOOD. It was not like a pizza. My husband and son specifically liked it BECAUSE it was not like pizza. It was more like a layered casserole. Will make it again!

Cold, or cooked … DH bought me one and I have a few recipes but am open to something different …

An old-fashioned dish that has recently become appealing in my mind, at least, again …

I modified a recipe, this is tonoght’s dessert:

3 eggs
1/3 c splenda
2 c coconut cream
2 TBS Vanilla whey powder
vanilla and orange extracts (or whatever), 1/2 tsp ea

Mix in blender, bake 2 hr in casserole or cups in a water bath at 300, cool and chill before serving. The original recipe recommends it for breakfast too!

How about that prescription preparation of omega-3’s marketed to lower triglycerides?

… think THEIR ads mention that decreasing dietary carbohydrate might have the same or better effect? 😀

I just read on another forum that someone got hold of a charge card (somehow via FB) and bought up gift cards … well that was not so big … but then I got a "chat" message from a friend, ADVERTISING a website for gift cards …

It was not my friend of course.

BEWARE. If you get one from "me," please, let me know!!!

The Almanac.net

Came up on my google alert for low-carb diabetic news Very nice — thought I would share.

Oops — staceyy?

http://www.aace.com/pub/pdf/Glycemic…lAlgorithm.pdf

Too liberal on A1c’s, for my tastes!!! 🙁 6.5 seems to be their magic number these days?

I had a bag of these from my Oriental grocery … it’s Korean but carries Chinese and Japanese stuff too and these are from Taiwan.

I used them in my Thai soup and while they were chewier than wheat or rice noodles, take a bit of getting used to, I was going to put in Tofu so they were standing in for it AND noodles, and we were pretty happy with it. Not quite as, well, odd, as the shirataki noodles!

I have been feeling sorry for myself and my recent lack of control … and poor access to insulin.

Yesterday I came across this film Rockhopper.tv – Programmes

Which was immediately perspective changing … ! Having traveled in several developing countries, including India, I know the truths contained here. Heck, if you have even seen Slumdog Millionaire, you will understand. Imagine being insulin-dependent, in a protein-poor place, and unable to obtain what you need to survive, let alone grow up or thrive, as a diabetic.

Ann Nutr Metab. 2010 Jan 4;56(1):74-79. [Epub ahead of print]
Examination of the Antiglycemic Properties of Vinegar in Healthy Adults.

Johnston CS, Steplewska I, Long CA, Harris LN, Ryals RH.

Nutrition Program, College of Nursing and Health Innovation, Arizona State University, Mesa, Ariz., USA.

Background: Vinegar reduces postprandial glycemia (PPG) in healthy adults. This study investigated the vinegar dosage (10 vs. 20 g), timing (during mealtime vs. 5 h before meal) and application (acetic acid as vinegar vs. neutralized salt) for reducing PPG. Methods: Four randomized crossover trials were conducted in adults (n = 9-10/trial) with type 2 diabetes (1 trial) or without diabetes (3 trials). All trials followed the same protocol: a standardized meal the evening prior to testing, an overnight fast (>10 h) and 2-hour glucose testing following consumption of a bagel and juice test meal (3 trials) or dextrose solution (1 trial). For each trial, PPG was compared between treatments using area-under-the-curve calculations 120 min after the meal. Results: Two teaspoons of vinegar ( approximately 10 g) effectively reduced PPG, and this effect was most pronounced when vinegar was ingested during mealtime as compared to 5 h before the meal. Vinegar did not alter PPG when ingested with monosaccharides, suggesting that the antiglycemic action of vinegar is related to the digestion of carbohydrates. Finally, sodium acetate did not alter PPG, indicating that acetate salts lack antiglycemic properties. Conclusions: The antiglycemic properties of vinegar are evident when small amounts of vinegar are ingested with meals composed of complex carbohydrates. In these situations, vinegar attenuated PPG by approximately 20% compared to placebo. Copyright © 2010 S. Karger AG, Basel.

I really have worked hard to modify my health habits.

I know I was not eating great — and at the time, I KNEW I was not eating great — and not working out. I also felt stressed and pressed for time. I ALSO have mitigating factors in my development of D — two now — GADAs, AND Niacin (apparently I am a reactor). And I knew I was becoming obese — teetering on the edge of it, but nonetheless. I also know my habits were being driven by my blood sugar, especially just pre-dx. Heck, I sought medical help 7 or 8 mos BEFORE diagnosis, and blood sugar was one thing that was checked.

So how do you deal with the people who just have to say, "TOLJA SO!" despite being human themselves, with health habit shortcomings? What do you say? It just seems childish. I suspect misery loves company, and this person wants me to blame myself for my condition.

PS this person did, and still DOES email me telling me about their junk-food fests.

Scientists link plastics chemical to health risks – Yahoo! News

… and, of course, Diabetes, again.

Endocr Pract. 2009 Nov 26:1-13. [Epub ahead of print]
Vitamin B12 Deficiency is Common in Subjects with Type 2 Diabetes Mellitus Not Taking Metformin and is Nutritional in Nature.

Jawa AA, Akram J, Sultan M, Humayoun A, Raza R.

Jinnah – Allama Iqbal Institute of Diabetes and Endocrinology, Allama Iqbal Medical College/Jinnah Hospital, Lahore, 54550, Pakistan.

OBJECTIVES: The primary objective of our study was to estimate the frequency of undiagnosed Vitamin B12 deficiency amongst subjects with type 2 diabetes who were not taking Metformin for at least the past 5 years. The secondary objective was to ascertain if Vitamin B12 deficiency amongst the type 2 diabetes subjects was due to nutritional deficiency or malabsorption. METHODS: Vitamin B12 levels were measured in 44 subjects with diabetes (40-70 years). 21/44 (48%) had low vitamin B12 levels (<200 microgram/dL). 10/21 subjects agreed to enter an intervention phase comprising oral mecobalamin 1500 microgram/day for 3 months. Those subjects who failed to normalize Vitamin B12 levels after oral supplementation alone would be presumed to have Vitamin B12 deficiency due to malabsorption. RESULTS: Almost one-half of type 2 diabetes subjects not taking Metformin had biochemically proven vitamin B12 deficiency. 10/10 subjects had normalized their Vitamin B12 levels after 3 months of oral supplementation. CONCLUSION: We conclude that Vitamin B12 deficiency is common amongst type 2 diabetes subjects and is nutritional in nature. In addition to intensive glycemic control, Vitamin B12 supplementation should also be considered for treatment of diabetic neuropathy. In almost 50% of low vitamin B12 subjects the deficiency was correctable with oral supplementation alone. This indeed is an important finding, as taking oral Vitamin B12 supplementation is easy, convenient and readily accepted by patients. This is a novel finding and stresses the need for aggressive and early diagnosis and treatment to avoid complications of Vitamin B12 deficiency.

😀 Do ya think it is related to their nutritional instruction, then … ?

Diabetes Metab. 2010 Jan 7. [Epub ahead of print]
Clinical and metabolic characteristics of patients with latent autoimmune diabetes in adults (LADA): Absence of rapid beta-cell loss in patients with tight metabolic control.

Chaillous L, Bouhanick B, Kerlan V, Mathieu E, Lecomte P, Ducluzeau PH, Delamaire M, Sonnet E, Maugendre D, Maréchaud R, Rohmer V, Saï P, Charbonnel B.

Clinique d’endocrinologie, maladies métaboliques et nutrition, institut du thorax, hôpital Laennec, boulevard Jacques-Monod, 44093 Nantes cedex 1, France; Laboratoire d’immuno-endocrinologie, ENVN, Atlanpôle-La-Chantrerie, BP 40706, 44307 Nantes cedex 3, France.

AIM AND METHODS: The present study compared the clinical and metabolic characteristics of latent autoimmune diabetes in adults (LADA) with type 2 diabetes, as well as the residual beta-cell function and progression to insulin treatment, over a 2-year follow-up period, of antibody (Ab)-positive and Ab-negative patients who achieved tight glycaemic control (HbA(1c) 7.0+/-0.8% and 6.5+/-0.9%, respectively, at the time of entry into the study). RESULTS: Glutamic acid decarboxylase antibodies (GADA) and/or islet cell antibodies (ICA) were detected in 10% of patients presenting with non-insulin-dependent diabetes. Around half of Ab-positive patients required insulin treatment during the follow-up. Ab-positive patients displayed lower stimulated C-peptide levels both at entry and during the follow-up compared with Ab-negative patients, although no significant decline in C-peptide levels was observed in either subgroup over two years. Nevertheless, Ab-positive patients progressed more frequently to insulin treatment, and stimulated C-peptide tended to decrease in LADA patients who subsequently required insulin, whereas it remained stable in those who were non-insulin-dependent. In those who progressed, the trend towards C-peptide decline persisted even after starting insulin treatment. CONCLUSION: LADA patients demonstrate lower residual beta-cell function than do type 2 diabetes patients. However, those who achieve tight metabolic control do not present with a rapid decline in beta-cell function. Further studies are needed to determine the optimal treatment strategy in such patients. Copyright © 2009 Elsevier Masson SAS. All rights reserved.

PMID: 20060765 [PubMed – as supplied by publisher]:D

The use of metformin in type 1 diabetes: a systema… [Diabetologia. 2010] – PubMed result

Hopefully this will be helpful!

Before dinner: 116

Dinner was: a 2 egg omelet with cheese (maybe 1.4 c) and a veggie sausage (6 g) and a bit of chile verde salsa (4 g max) … so I called it 12 g. And 2 tiny squares of G&B chocolate, so I called those 2 g? So if I am not kidding myself, call dinner 14 – 16 g carbs, total.

After dinner, 30 min exercycle workout, with intervals, and cycle said 120 calories.

After workout, 3 hr, postprandial: 114.

Should I be VENTING???? Because I want to!!!! @#$#%$%&*:mad: 🙁

Worse yet, same workout, I was still taking niacin, dropped me 50 pts last week. NOT predictable, at ALL!

Anyone read it yet? Or seen reviews? It looks interesting!

My adventure with Niacin continues … I lowered my dose to the starting amount (500 per day, divided) and plotted my blood sugars and the effects of ramping up and lowering were evident, indeed.

In the meantime I had begun exercising, so was feeling pretty optimistic about my ability to handle taking it.

This afternoon I had a snack of yogurt and 1/3 cup blueberries, and went off and had a nap. Not giving the blueberries a second thought … (they have spiked me in the past).

At 5 pm I felt a bit hungry so decided to test — 160. And re-test, 152. My poor husband was on the phone with me at the time!

I did 30 minutes workout on my exbike … and 40 minutes later, re-tested … at 76! And no, oddly, I did NOT feel any discomfort from that rapid of a blip … went on and ate my dinner, gonna do another minutes now.

Postprandial dysmetabolism, or postprandial dyslipidemia.

Coined by a cardiologist local to me, some interesting articles come up.

The Relevance of C-Peptide in Diabetes and its Complications: An Introduction to the Special Issue.

Sima AA, Wahren J.

Rev Diabet Stud. 2009 Fall;6(3):136-137. Epub 2009 Nov 10.PMID: 20039002 [PubMed – as supplied by publisher]

Okay, I cannot access this title, but anyone interested, might care to see if you can!? The articles examine C-peptide’s role in various complications of D.